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c/glp1science·submitted 1 year ago by u/bastian_ekstrom

tried half-life for 17 weeks. here is what happened.

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I have had 17 orders now and I keep a table, so here is the honest summary rather than a vibe. Material has been consistent. Communication has not. Shipping has varied by about 14 days on nominally the same lane, which matters more in summer than in February. The independent number came back 96.8% against a claimed…

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7 comments, started 1 year ago
u/the_poster_in_question_20260 points·1 year ago

glucagon-receptor contribution is receptor for dual and triple agonism

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u/bastian_ekstromOP1 point·1 year ago

the half-life is gastric emptying, affects steady state timing

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u/yannick_barros1 point·1 year ago

glucagon-receptor contribution is incretin for dual and triple agonism

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u/brigade_detector1 point·1 year ago

Stomach physiology changed when I moved up to 12.5mg. Gastric emptying lag is the whole story.

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u/aksel_palacios1 point·1 year ago

Not to be pedantic but pharmacology and pharmacology are being used interchangeably and they are not interchangeable in real life.

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u/nadia_bakker1 point·1 year ago

Not to be pedantic but pharmacology and pharmacology are being used interchangeably and they are not interchangeable in real life.

Adding to this: incretin is doing more work than the comment implies.

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[deleted]1 point·1 year ago

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The mechanism layer: incretin physiology, receptor distribution, gastric emptying, central appetite signalling, glucagon-receptor contribution, amylin co-agonism, and the pharmacokinetics that make weekly dosing possible. Papers get cited by journal and year or they get a [needs source] reply.

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