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c/glp1science·submitted 1 year ago by u/cato_batista

why does nobody talk about appetite

Speculationbranch of 5 comments

Confession thread, sort of. I went from 2.4mg to 0.5mg much faster than I should have because the scale had stalled and I got impatient. The stall broke about 2 weeks later, at which point I had no way of knowing whether the dose increase did anything or whether it would have broken anyway. So now I have constipation…

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5 comments, started 1 year ago
u/rina_bergstrom37 points·1 year ago

Rodent data is rodent data. Dose scaling is not linear and the models tell you what to investigate, not what to expect.

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u/rasmus_kimani24 points·1 year ago

rodent appetite tells you what to investigate, not what to expect in humans

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u/cato_batistaOP18 points·1 year ago

Receptor distribution explains why fatigue hits receptor but not pharmacology.

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u/marisol_kravchenko6 points·1 year ago

Not to be pedantic but gastric emptying and receptor are being used interchangeably and they are not interchangeable in real life.

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u/the_poster_in_question_20269 points·1 year ago

do not extrapolate rodent data to human dosing without saying so

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The mechanism layer: incretin physiology, receptor distribution, gastric emptying, central appetite signalling, glucagon-receptor contribution, amylin co-agonism, and the pharmacokinetics that make weekly dosing possible. Papers get cited by journal and year or they get a [needs source] reply.

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