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Single comment threadYou are looking at one branch of genuine question about amylin that I am slightly embarrassed to ask — 57 comments in the full submission. View in context.
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c/cagrilintide·submitted 1 year ago by u/pancreatitis_scare

genuine question about amylin that I am slightly embarrassed to ask

Trial Databranch of 8 comments

Something I noticed reading old threads that I have not seen said out loud. The advice on REDEFINE in this community changed substantially around the start of last year, and nobody went back and updated the older posts. So depending on which thread the search engine hands you, you get two contradictory answers with…

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8 comments, started 1 year ago
u/niels_norgaard41 points·1 year ago

Yeah, amylin is doing different work than GLP-1.

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u/nl_verzekering10 points·1 year ago

Are we talking amylin or GLP-1 here?

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u/noor_hovland3 points·1 year ago

the co-agonism question is real and interesting

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u/ilias_kaufmann14 points·1 year ago

Yeah, amylin is doing different work than GLP-1.

Adding to this: co-agonism is doing more work than the comment implies.

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u/zeynep_weiss8 points·1 year ago

not approved anywhere, keep posts descriptive, not instructional

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u/helga_vermeulen5 points·1 year ago

amylin receptor distribution is different from GLP-1

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u/yannick_salinas4 points·1 year ago

the amylin agonist pool is small, community is satiety

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u/crosspost_bot_no10 points·1 year ago

the mechanism is different, the side effect profile reflects it

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Amylin analog pharmacology and the CagriSema combination: why amylin and GLP-1 co-agonism produces a different satiety profile, the REDEFINE readouts, and the very limited pool of people handling cagrilintide as research material.

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