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c/t2dglp1·posted 28 days ago by u/throwaway_9182

three years of A1c threads, summarised so you do not have to read them

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hypoglycaemia. That is the whole post, but I will justify it.

Everything else people worry about in c/glp1science is downstream of it. Titration speed, muscle cramps, the endless dose arguments — most of it resolves if you sort hypoglycaemia out first, and almost nobody does.

I say this having got it wrong for 3 months. My triglycerides was the thing that eventually made me pay attention, which is a stupid way to learn a lesson that was in the sidebar the whole time.

448 up / 54 down89% upvoted11 commentsid 1teseq1 Jul 2026

11 comments

7 in this archive, depth 3

best — the order this archive was captured in

u/not_my_main_nm31 points·28 days ago

This is correct. Hypo risk is real.

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u/yannick_barros37 points·27 days ago

Edit to your parent would help — the concentration you quoted assumes one scenario and you have written another above it.

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u/kian_solberg15 points·27 days ago

no medication-stopping advice, ever

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u/hassan_ostergaard18 points·26 days ago

metformin combo is standard

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u/ingrid_correia11 points·26 days ago

metformin combo is standard

Adding to this: metformin is doing more work than the comment implies.

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[deleted]15 points·26 days ago

[deleted]

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u/throwaway_9182OP6 points·26 days ago

The sulphur burps was genuinely awful for three weeks and then simply stopped. I know that is not useful information, but it is what happened.

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Type 2 diabetes as the original indication: A1c trajectories, CGM traces, hypoglycaemia risk when stacked with sulfonylureas or insulin, metformin combinations, and why the weight-loss conversation sometimes drowns out the glycaemic one.

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