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c/retatrutide·submitted 1 year ago by u/yannick_barros

my A1c moved and I cannot work out whether phase 2 is why

Trial Databranch of 8 comments

Long-ish post, sorry. tl;dr at the bottom. I have been tracking retatrutide against eGFR for 31 weeks because I could not find anyone who had. The correlation is weaker than I expected, which is itself mildly interesting given how confidently people link the two in here. Caveats up front: one person, one lab, one…

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This branch

8 comments, started 1 year ago
u/halima_mbeki152 points·1 year ago

This is correct. Triple agonism is genuinely different.

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u/rui_only_ro55 points·1 year ago

The glucagon component does different things than dual agonism. The nausea are not just amplified versions.

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u/kofi_balogun16 points·1 year ago

the dosing is steep, the sulphur burps reports are real, and nobody knows the human safety yet

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u/yannick_barrosOP0 points·1 year ago

the 7.5mg arm is different from 5mg, results do not transfer

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u/freya_baptista1 point·1 year ago

the weight curves are unusually steep, which makes predicting individual response harder

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u/lurker_for_years1 point·1 year ago·edited

nobody should be giving this to anyone without extensive monitoring

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u/two_mil_or_one1 point·1 year ago

phase 2 numbers are not maintenance numbers, cite the trial

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u/halima_mbeki1 point·1 year ago

triple agonism is glucagon, do not generalise

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About c/retatrutide

Retatrutide: GIP/GLP-1/glucagon triple agonism, the TRIUMPH programme, and the fact that most people discussing it are handling research-grade material with no human-use approval anywhere. Dose-response, heart-rate signal, and the unusually steep phase-2 weight curves get argued about here weekly.

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