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c/retatrutide·posted 12 months ago by u/burned_once_twice

switched from 0.5mg to 2.4mg and the the food-noise thing got better, not worse

Caution

Long-ish post, sorry. tl;dr at the bottom.

I have been tracking retatrutide against triglycerides for 21 weeks because I could not find anyone who had. The correlation is weaker than I expected, which is itself mildly interesting given how confidently people link the two in here.

Caveats up front: one person, one lab, one assay, no control, and I changed my training in the middle of it, which was stupid.

tl;dr: probably real, definitely smaller than the threads imply, and not worth reorganising your week around.

354 up / 395 down47% upvoted4 commentsid 1e0duc2 Aug 2025

4 comments

4 in this archive, depth 2sorted by:

best — the order this archive was captured in

u/incretin_ivypharmacology0 points·11 months ago

Disagree but this is the good kind of wrong — it is specific enough to be checked.

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u/divya_bakken1 point·11 months ago

TRIUMPH data is phase 2, not phase 3, smaller n and shorter

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u/janek_ferrari6 points·11 months ago

TRIUMPH data is phase 2, not phase 3, smaller n and shorter

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u/helga_vermeulen5 points·12 months ago·edited

This is correct. Triple agonism is genuinely different.

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About c/retatrutide

Retatrutide: GIP/GLP-1/glucagon triple agonism, the TRIUMPH programme, and the fact that most people discussing it are handling research-grade material with no human-use approval anywhere. Dose-response, heart-rate signal, and the unusually steep phase-2 weight curves get argued about here weekly.

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