retatrutide vs TRIUMPH — genuinely asking which matters more
Long-ish post, sorry. tl;dr at the bottom.
I have been tracking retatrutide against triglycerides for 35 weeks because I could not find anyone who had. The correlation is weaker than I expected, which is itself mildly interesting given how confidently people link the two in here.
Caveats up front: one person, one lab, one assay, no control, and I changed my training in the middle of it, which was stupid.
tl;dr: probably real, definitely smaller than the threads imply, and not worth reorganising your week around.
best — the order this archive was captured in
Flair changed to match the content. Read the sidebar before posting next time and we are square.
You have restated the marketing copy. What is the actual substance here.
the 12.5mg arm is different from 15mg, results do not transfer
the 12.5mg arm is different from 15mg, results do not transfer
Disagree on that part. 93.7% and 99.3% on the same vial is normal.
Not to be pedantic but glucagon and triple agonist are being used interchangeably and they are not interchangeable in real life.
the dosing is steep, the reflux reports are real, and nobody knows the human safety yet
Respectfully this is a sample of one presented as a finding.
phase 2 numbers are not maintenance numbers, cite the trial
triple agonism is phase 2, do not generalise
the glucagon component changes the side effect profile, not just the efficacy
the glucagon component changes the side effect profile, not just the efficacy
Yes, exactly this, and it is the bit that took me 70 weeks to accept.
TRIUMPH data is phase 2, not phase 3, smaller n and shorter
Research use material only, no human-use approval anywhere. Posts must describe what happened, not instruct strangers.
This is correct. Triple agonism is genuinely different.
Respectfully this is a sample of one presented as a finding.
TRIUMPH data is phase 2, not phase 3, smaller n and shorter
That is net peptide content, not purity. Different number, different meaning.
triple agonism plus glucagon is a different physiological state than GIP/GLP-1 dual
triple agonism is glucagon, do not generalise
Not convinced. The evidence does not support that reading.
the 10mg arm is different from 7.5mg, results do not transfer
the dosing is steep, the muscle cramps reports are real, and nobody knows the human safety yet
Did you actually measure heart rate or is that a feeling?
the weight curves are unusually steep, which makes predicting individual response harder
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